Contribution of Heptose Metabolites and the cag Pathogenicity Island to the Activation of Monocytes/Macrophages by Helicobacter pylori

Eitemau Tebyg

• Helicobacter pylori modulates host cell responses by CagT4SS-dependent translocation of an intermediate metabolite of LPS inner core heptose biosynthesis.
  gan: Saskia C Stein, et al.
  Cyhoeddwyd: (2017-07-01)
• Helicobacter pylori modulates host cell responses by CagT4SS-dependent translocation of an intermediate metabolite of LPS inner core heptose biosynthesis
  gan: Stein, Saskia C., et al.
  Cyhoeddwyd: (2017)
• The Helicobacter pylori cag Pathogenicity Island
  gan: Noto, Jennifer M., et al.
  Cyhoeddwyd: (2012)
• Helicobacter pylori CagT4SS proteins CagN and CagM bind DNA and CagN is involved in heptose-independent pro-inflammatory substrate translocation by the T4SS
  gan: Simon H. Bats, et al.
  Cyhoeddwyd: (2025-09-01)
• CagA, the cag pathogenicity island and Helicobacter pylori virulence
  gan: ATHERTON, J
  Cyhoeddwyd: (1999)