טוען...
TRPC1- and TRPC3-dependent Ca(2+) signaling in mouse cortical astrocytes affects injury-evoked astrogliosis in vivo
Following brain injury astrocytes change into a reactive state, proliferate and grow into the site of lesion, a process called astrogliosis, initiated and regulated by changes in cytoplasmic Ca(2+). Transient receptor potential canonical (TRPC) channels may contribute to Ca(2+) influx but their pres...
שמור ב:
| הוצא לאור ב: | Glia |
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| Main Authors: | , , , , , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2017
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5526095/ https://ncbi.nlm.nih.gov/pubmed/28636132 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/glia.23180 |
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