TRPC1- and TRPC3-dependent Ca(2+) signaling in mouse cortical astrocytes affects injury-evoked astrogliosis in vivo

Following brain injury astrocytes change into a reactive state, proliferate and grow into the site of lesion, a process called astrogliosis, initiated and regulated by changes in cytoplasmic Ca(2+). Transient receptor potential canonical (TRPC) channels may contribute to Ca(2+) influx but their pres...

Cijeli opis

Spremljeno u:
Bibliografski detalji
Izdano u:Glia
Glavni autori: Belkacemi, Thabet, Niermann, Alexander, Hofmann, Laura, Wissenbach, Ulrich, Birnbaumer, Lutz, Leidinger, Petra, Backes, Christina, Meese, Eckart, Keller, Andreas, Bai, Xianshu, Scheller, Anja, Kirchhoff, Frank, Philipp, Stephan E., Weissgerber, Petra, Flockerzi, Veit, Beck, Andreas
Format: Artigo
Jezik:Inglês
Izdano: 2017
Teme:
Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5526095/
https://ncbi.nlm.nih.gov/pubmed/28636132
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/glia.23180
Oznake: Dodaj oznaku
Bez oznaka, Budi prvi tko označuje ovaj zapis!

Similar Items