TRPC1- and TRPC3-dependent Ca(2+) signaling in mouse cortical astrocytes affects injury-evoked astrogliosis in vivo

Following brain injury astrocytes change into a reactive state, proliferate and grow into the site of lesion, a process called astrogliosis, initiated and regulated by changes in cytoplasmic Ca(2+). Transient receptor potential canonical (TRPC) channels may contribute to Ca(2+) influx but their pres...

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Vydáno v:Glia
Hlavní autoři: Belkacemi, Thabet, Niermann, Alexander, Hofmann, Laura, Wissenbach, Ulrich, Birnbaumer, Lutz, Leidinger, Petra, Backes, Christina, Meese, Eckart, Keller, Andreas, Bai, Xianshu, Scheller, Anja, Kirchhoff, Frank, Philipp, Stephan E., Weissgerber, Petra, Flockerzi, Veit, Beck, Andreas
Médium: Artigo
Jazyk:Inglês
Vydáno: 2017
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5526095/
https://ncbi.nlm.nih.gov/pubmed/28636132
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/glia.23180
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