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Constitutive increases in corticotropin releasing factor and fatty acid amide hydrolase drive an anxious phenotype
BACKGROUND: Corticotropin-releasing factor (CRF) mediates anxiogenic responses by activating CRF(1) receptors in limbic brain regions. Anxiety is further modulated by the endogenous cannabinoid (eCB) system that attenuates the synaptic effects of stress. In the amygdala, acute stress activates the e...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Biol Psychiatry |
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| Κύριοι συγγραφείς: | , , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2017
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5509512/ https://ncbi.nlm.nih.gov/pubmed/28209423 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.biopsych.2017.01.005 |
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