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Caveolin‐1 deficiency induces premature senescence with mitochondrial dysfunction
Paradoxical observations have been made regarding the role of caveolin‐1 (Cav‐1) during cellular senescence. For example, caveolin‐1 deficiency prevents reactive oxygen species‐induced cellular senescence despite mitochondrial dysfunction, which leads to senescence. To resolve this paradox, we re‐ad...
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| Publicado no: | Aging Cell |
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| Main Authors: | , , , , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5506423/ https://ncbi.nlm.nih.gov/pubmed/28514055 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12606 |
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