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Fibroblast deletion of ROCK2 attenuates cardiac hypertrophy, fibrosis, and diastolic dysfunction

Although left ventricular (LV) diastolic dysfunction is often associated with hypertension, little is known regarding its underlying pathophysiological mechanism. Here, we show that the actin cytoskeletal regulator, Rho-associated coiled-coil containing kinase-2 (ROCK2), is a critical mediator of LV...

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Pubblicato in:JCI Insight
Autori principali: Shimizu, Toru, Narang, Nikhil, Chen, Phetcharat, Yu, Brian, Knapp, Maura, Janardanan, Jyothi, Blair, John, Liao, James K.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Clinical Investigation 2017
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5499369/
https://ncbi.nlm.nih.gov/pubmed/28679962
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.93187
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