Pharmacologic targeting ERK1/2 attenuates the development and progression of hyperuricemic nephropathy in rats

The pathogenesis of hyperuricemia-induced chronic kidney disease is largely unknown. In this study, we investigated whether extracellular signal–regulated kinases1/2 (ERK1/2) would contribute to the development of hyperuricemic nephropathy (HN). In a rat model of HN induced by feeding mixture of ade...

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Bibliografiske detaljer
Udgivet i:Oncotarget
Main Authors: Liu, Na, Xu, Liuqing, Shi, Yingfeng, Fang, Lu, Gu, Hongwei, Wang, Hongrui, Ding, Xiaoqiang, Zhuang, Shougang
Format: Artigo
Sprog:Inglês
Udgivet: Impact Journals LLC 2017
Fag:
Research Paper
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5464913/
https://ncbi.nlm.nih.gov/pubmed/28442634
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.16995
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