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Delayed treatment with an autophagy inhibitor 3-MA alleviates the progression of hyperuricemic nephropathy
Autophagy is a cell self-renewal process that relies on the degradation of the cytoplasmic proteins or organelles of lysosomes and is associated with development of numerous diseases. However, the therapeutic effect of autophagy inhibition on hyperuricemic nephropathy (HN) and the underlying mechani...
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| Publicat a: | Cell Death Dis |
|---|---|
| Autors principals: | , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Nature Publishing Group UK
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7298642/ https://ncbi.nlm.nih.gov/pubmed/32555189 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-020-2673-z |
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