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Enhanced proteostasis in post-ischemic stroke mouse brains by ubiquilin-1 promotes functional recovery
Stroke is pathologically associated with oxidative stress, protein damage, and neuronal loss. We previously reported that overexpression of a ubiquitin-like protein, ubiquilin-1 (Ubqln), protects neurons against ischemia-caused brain injury, while knockout of the gene exacerbates cerebral ischemia-c...
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| Vydáno v: | Cell Mol Neurobiol |
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| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5462886/ https://ncbi.nlm.nih.gov/pubmed/27928652 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10571-016-0451-3 |
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