Hyper-activation of pp60(Src) limits nitric oxide signaling by increasing asymmetric dimethylarginine levels during acute lung injury

The molecular mechanisms by which the endothelial barrier becomes compromised during lipopolysaccharide (LPS) mediated acute lung injury (ALI) are still unresolved. We have previously reported that the disruption of the endothelial barrier is due, at least in part, to the uncoupling of endothelial n...

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Publicado no:Free Radic Biol Med
Main Authors: Kumar, Sanjiv, Sun, Xutong, Noonepalle, Satish Kumar, Lu, Qing, Zemskov, Evgeny, Wang, Ting, Aggarwal, Saurabh, Gross, Christine, Sharma, Shruti, Desai, Ankit A, Hou, Yali, Dasarathy, Sridevi, Qu, Ning, Reddy, Vijay, Lee, Sung Gon, Cherian-Shaw, Mary, Yuan, Jason X.-J., Catravas, John D., Rafikov, Ruslan, Garcia, Joe G.N., Black, Stephen M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5449193/
https://ncbi.nlm.nih.gov/pubmed/27838434
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2016.11.008
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