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GIRK2 splice variants and neuronal G protein-gated K(+) channels: implications for channel function and behavior
Many neurotransmitters directly inhibit neurons by activating G protein-gated inwardly rectifying K(+) (GIRK) channels, thereby moderating the influence of excitatory input on neuronal excitability. While most neuronal GIRK channels are formed by GIRK1 and GIRK2 subunits, distinct GIRK2 isoforms gen...
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| Pubblicato in: | Sci Rep |
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| Autori principali: | , , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Nature Publishing Group UK
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5431628/ https://ncbi.nlm.nih.gov/pubmed/28487514 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-017-01820-2 |
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