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Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction
Coronary collateral growth (CCG) is impaired in metabolic syndrome (MetS). microRNA-145 (miR-145-Adv) delivery to our rat model of MetS (JCR) completely restored and neutrophil depletion significantly improved CCG. We determined whether low endogenous levels of miR-145 in MetS allowed for elevated p...
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| Vydáno v: | Am J Physiol Heart Circ Physiol |
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| Hlavní autoři: | , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Physiological Society
2017
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5402017/ https://ncbi.nlm.nih.gov/pubmed/28011587 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00561.2016 |
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