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Induction of ACE and activation of the RAS contribute to 20-HETE-mediated endothelial dysfunction

OBJECTIVE: 20-HETE promotes endothelial dysfunction by uncoupling eNOS, stimulating O(2)(−) production and reducing NO bioavailability. Moreover, 20-HETE-dependent vascular dysfunction and hypertension are associated with upregulation of the renin-angiotensin system (RAS). This study was undertaken...

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Detalhes bibliográficos
Main Authors: Cheng, Jennifer, Garcia, Victor, Ding, Yan, Wu, Cheng-Chia, Thakar, Krutanjali, Falck, John R., Ramu, Errabelli, Schwartzman, Michal Laniado
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3418056/
https://ncbi.nlm.nih.gov/pubmed/22723444
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.112.248344
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