טוען...
Induction of ACE and activation of the RAS contribute to 20-HETE-mediated endothelial dysfunction
OBJECTIVE: 20-HETE promotes endothelial dysfunction by uncoupling eNOS, stimulating O(2)(−) production and reducing NO bioavailability. Moreover, 20-HETE-dependent vascular dysfunction and hypertension are associated with upregulation of the renin-angiotensin system (RAS). This study was undertaken...
שמור ב:
| Main Authors: | , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2012
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3418056/ https://ncbi.nlm.nih.gov/pubmed/22723444 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.112.248344 |
| תגים: |
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