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Surface-modified particles loaded with CaMKII inhibitor protect cardiac cells against mitochondrial injury
An excess of calcium (Ca(2+)) influx into mitochondria during mitochondrial re-energization is one of the causes of myocardial cell death during ischemic/reperfusion injury. This overload of Ca(2+) triggers the mitochondrial permeability transition pore (mPTP) opening which leads to programmed cell...
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| Publicado no: | Int J Pharm |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5401631/ https://ncbi.nlm.nih.gov/pubmed/28167264 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ijpharm.2017.01.061 |
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