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BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silicosis
Following inhalation into the lungs, silica particles are engulfed by alveolar macrophages, which triggers endogenous or exogenous apoptosis signaling pathways. As an inducer of apoptosis, the role of BBC3/PUMA (BCL2-binding component 3) in macrophages during silicosis remains unknown. Here, we expo...
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| Published in: | Cell Death Dis |
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| Main Authors: | , , , , , , , , , , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
Nature Publishing Group
2017
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5386570/ https://ncbi.nlm.nih.gov/pubmed/28277537 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2017.78 |
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