BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silicosis

Following inhalation into the lungs, silica particles are engulfed by alveolar macrophages, which triggers endogenous or exogenous apoptosis signaling pathways. As an inducer of apoptosis, the role of BBC3/PUMA (BCL2-binding component 3) in macrophages during silicosis remains unknown. Here, we expo...

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Udgivet i:Cell Death Dis
Main Authors: Liu, Haijun, Cheng, Yusi, Yang, Jian, Wang, Wei, Fang, Shencun, Zhang, Wei, Han, Bing, Zhou, Zewei, Yao, Honghong, Chao, Jie, Liao, Hong
Format: Artigo
Sprog:Inglês
Udgivet: Nature Publishing Group 2017
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Original Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5386570/
https://ncbi.nlm.nih.gov/pubmed/28277537
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2017.78
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