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circHECTD1 promotes the silica-induced pulmonary endothelial–mesenchymal transition via HECTD1
Excessive proliferation and migration of fibroblasts contribute to pulmonary fibrosis in silicosis, and both epithelial cells and endothelial cells participate in the accumulation of fibroblasts via the epithelial–mesenchymal transition (EMT) and the endothelial–mesenchymal transition (EndMT), respe...
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| Pubblicato in: | Cell Death Dis |
|---|---|
| Autori principali: | , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Nature Publishing Group UK
2018
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5852113/ https://ncbi.nlm.nih.gov/pubmed/29540674 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-018-0432-1 |
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