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circHECTD1 promotes the silica-induced pulmonary endothelial–mesenchymal transition via HECTD1

Excessive proliferation and migration of fibroblasts contribute to pulmonary fibrosis in silicosis, and both epithelial cells and endothelial cells participate in the accumulation of fibroblasts via the epithelial–mesenchymal transition (EMT) and the endothelial–mesenchymal transition (EndMT), respe...

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Bibliografski detalji
Izdano u:Cell Death Dis
Glavni autori: Fang, Shencun, Guo, Huifang, Cheng, Yusi, Zhou, Zewei, Zhang, Wei, Han, Bing, Luo, Wei, Wang, Jing, Xie, Weiping, Chao, Jie
Format: Artigo
Jezik:Inglês
Izdano: Nature Publishing Group UK 2018
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5852113/
https://ncbi.nlm.nih.gov/pubmed/29540674
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-018-0432-1
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