Synaptic Mitochondria Sustain More Damage than Non-Synaptic Mitochondria after Traumatic Brain Injury and Are Protected by Cyclosporine A

Currently, there are no Food and Drug Administration (FDA)-approved pharmacotherapies for the treatment of those with traumatic brain injury (TBI). As central mediators of the secondary injury cascade, mitochondria are promising therapeutic targets for prevention of cellular death and dysfunction af...

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Detalhes bibliográficos
Publicado no:J Neurotrauma
Main Authors: Kulbe, Jacqueline R., Hill, Rachel L., Singh, Indrapal N., Wang, Juan A., Hall, Edward D.
Formato: Artigo
Idioma:Inglês
Publicado em: Mary Ann Liebert, Inc. 2017
Assuntos:
Original Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5385586/
https://ncbi.nlm.nih.gov/pubmed/27596283
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2016.4628
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