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Inhibition of c-Jun NH(2)-terminal kinase or extracellular signal-regulated kinase improves lung injury
BACKGROUND: Although in vitro studies have determined that the activation of mitogen-activated protein (MAP) kinases is crucial to the activation of transcription factors and regulation of the production of proinflammatory mediators, the roles of c-Jun NH(2)-terminal kinase (JNK) and extracellular s...
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| Pubblicato in: | Respir Res |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
BMC
2004
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC538282/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15566575/ https://ncbi.nlm.nih.govhttps://doi.org/10.1186/1465-9921-5-23 |
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