Inhibition of c-Jun NH(2)-terminal kinase or extracellular signal-regulated kinase improves lung injury

BACKGROUND: Although in vitro studies have determined that the activation of mitogen-activated protein (MAP) kinases is crucial to the activation of transcription factors and regulation of the production of proinflammatory mediators, the roles of c-Jun NH(2)-terminal kinase (JNK) and extracellular s...

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Publicado en:Respir Res
Autores principales: Lee, Hui Su, Kim, Hee Jae, Moon, Chang Sook, Chong, Young Hae, Kang, Jihee Lee
Formato: Artigo
Lenguaje:Inglês
Publicado: BMC 2004
Materias:
Research
Acceso en línea:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC538282/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15566575/
https://ncbi.nlm.nih.govhttps://doi.org/10.1186/1465-9921-5-23
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