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Inhibition of c-Jun NH(2)-terminal kinase or extracellular signal-regulated kinase improves lung injury
BACKGROUND: Although in vitro studies have determined that the activation of mitogen-activated protein (MAP) kinases is crucial to the activation of transcription factors and regulation of the production of proinflammatory mediators, the roles of c-Jun NH(2)-terminal kinase (JNK) and extracellular s...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2004
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC538282/ https://ncbi.nlm.nih.gov/pubmed/15566575 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1465-9921-5-23 |
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