Retention of normal glia function by an isoform-selective protein kinase inhibitor drug candidate that modulates cytokine production and cognitive outcomes

BACKGROUND: Brain p38α mitogen-activated protein kinase (MAPK), a potential therapeutic target for cognitive dysfunction based on the neuroinflammation-synaptic dysfunction cycle of pathophysiology progression, offers an innovative pharmacological strategy via inhibiting the same activated target in...

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Detalhes bibliográficos
Publicado no:J Neuroinflammation
Main Authors: Zhou, Zhengqiu, Bachstetter, Adam D., Späni, Claudia B., Roy, Saktimayee M., Watterson, D. Martin, Van Eldik, Linda J.
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2017
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5382362/
https://ncbi.nlm.nih.gov/pubmed/28381303
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-017-0845-2
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