Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells

Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...

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Detaylı Bibliyografya
Yayımlandı:Am J Physiol Renal Physiol
Asıl Yazarlar: Wu, Min, Zhang, Jian-Dong, Tang, Ri-Ning, Crowley, Steven D., Liu, Hong, Lv, Lin-Li, Ma, Kun-Ling, Liu, Bi-Cheng
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2016
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/
https://ncbi.nlm.nih.gov/pubmed/27582099
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016
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