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Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells
Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...
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| Pubblicato in: | Am J Physiol Renal Physiol |
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| Autori principali: | , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Physiological Society
2016
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/ https://ncbi.nlm.nih.gov/pubmed/27582099 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016 |
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