Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells

Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...

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Bibliografski detalji
Izdano u:Am J Physiol Renal Physiol
Glavni autori: Wu, Min, Zhang, Jian-Dong, Tang, Ri-Ning, Crowley, Steven D., Liu, Hong, Lv, Lin-Li, Ma, Kun-Ling, Liu, Bi-Cheng
Format: Artigo
Jezik:Inglês
Izdano: American Physiological Society 2016
Teme:
Research Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/
https://ncbi.nlm.nih.gov/pubmed/27582099
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016
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