Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells

Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...

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Detalhes bibliográficos
Publicado no:Am J Physiol Renal Physiol
Main Authors: Wu, Min, Zhang, Jian-Dong, Tang, Ri-Ning, Crowley, Steven D., Liu, Hong, Lv, Lin-Li, Ma, Kun-Ling, Liu, Bi-Cheng
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2016
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/
https://ncbi.nlm.nih.gov/pubmed/27582099
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016
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