Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells

Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...

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Enregistré dans:
Détails bibliographiques
Publié dans:Am J Physiol Renal Physiol
Auteurs principaux: Wu, Min, Zhang, Jian-Dong, Tang, Ri-Ning, Crowley, Steven D., Liu, Hong, Lv, Lin-Li, Ma, Kun-Ling, Liu, Bi-Cheng
Format: Artigo
Langue:Inglês
Publié: American Physiological Society 2016
Sujets:
Research Article
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/
https://ncbi.nlm.nih.gov/pubmed/27582099
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016
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