Elevated PTH induces endothelial-to-chondrogenic transition in aortic endothelial cells

Previous studies have shown that increased parathyroid hormone (PTH) attributable to secondary hyperparathyroidism in chronic kidney disease accelerates the arteriosclerotic fibrosis and calcification. Although the underlying mechanisms remain largely unknown, endothelial cells (ECs) have recently b...

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Vydáno v:Am J Physiol Renal Physiol
Hlavní autoři: Wu, Min, Zhang, Jian-Dong, Tang, Ri-Ning, Crowley, Steven D., Liu, Hong, Lv, Lin-Li, Ma, Kun-Ling, Liu, Bi-Cheng
Médium: Artigo
Jazyk:Inglês
Vydáno: American Physiological Society 2016
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Research Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5374307/
https://ncbi.nlm.nih.gov/pubmed/27582099
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00210.2016
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