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Deletion of the Fractalkine Receptor, CX3CR1, Improves Endogenous Repair, Axon Sprouting, and Synaptogenesis after Spinal Cord Injury in Mice

Impaired signaling via CX3CR1, the fractalkine receptor, promotes recovery after traumatic spinal contusion injury in mice, a benefit achieved in part by reducing macrophage-mediated injury at the lesion epicenter. Here, we tested the hypothesis that CX3CR1-dependent changes in microglia and macroph...

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Bibliografiset tiedot
Julkaisussa:J Neurosci
Päätekijät: Freria, Camila M., Hall, Jodie C.E., Wei, Ping, Guan, Zhen, McTigue, Dana M., Popovich, Phillip G.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2017
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5373135/
https://ncbi.nlm.nih.gov/pubmed/28264978
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2841-16.2017
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