The potassium channel KCa3.1 constitutes a pharmacological target for neuroinflammation associated with ischemia/reperfusion stroke

Activated microglia/macrophages significantly contribute to the secondary inflammatory damage in ischemic stroke. Cultured neonatal microglia express the K(+) channels Kv1.3 and KCa3.1, both of which have been reported to be involved in microglia-mediated neuronal killing, oxidative burst and cytoki...

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Detaylı Bibliyografya
Yayımlandı:J Cereb Blood Flow Metab
Asıl Yazarlar: Chen, Yi-Je, Nguyen, Hai M, Maezawa, Izumi, Grössinger, Eva M, Garing, April L, Köhler, Ralf, Jin, Lee-Way, Wulff, Heike
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: SAGE Publications 2015
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5363659/
https://ncbi.nlm.nih.gov/pubmed/26661208
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X15611434
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