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Angiotensin II induces kidney inflammatory injury and fibrosis through binding to myeloid differentiation protein-2 (MD2)

Growing evidence indicates that angiotensin II (Ang II), a potent biologically active product of RAS, is a key regulator of renal inflammation and fibrosis. In this study, we tested the hypothesis that Ang II induces renal inflammatory injury and fibrosis through interaction with myeloid differentia...

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Bibliografiska uppgifter
I publikationen:Sci Rep
Huvudupphovsmän: Xu, Zheng, Li, Weixin, Han, Jibo, Zou, Chunpeng, Huang, Weijian, Yu, Weihui, Shan, Xiaoou, Lum, Hazel, Li, Xiaokun, Liang, Guang
Materialtyp: Artigo
Språk:Inglês
Publicerad: Nature Publishing Group 2017
Ämnen:
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5359637/
https://ncbi.nlm.nih.gov/pubmed/28322341
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep44911
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