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Zika virus evades interferon-mediated antiviral response through the co-operation of multiple nonstructural proteins in vitro
Type I interferon (IFN) serves as the first line of defense against invading pathogens. Inhibition of IFN-triggered signaling cascade by Zika virus (ZIKV) plays a critical role for ZIKV to evade antiviral responses from host cells. Here we demonstrate that ZIKV nonstructural proteins NS1, NS4B and N...
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| 出版年: | Cell Discov |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group
2017
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5359216/ https://ncbi.nlm.nih.gov/pubmed/28373913 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/celldisc.2017.6 |
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