טוען...
Store-operated Ca(2+) entry controls ameloblast cell function and enamel development
Loss-of-function mutations in stromal interaction molecule 1 (STIM1) impair the activation of Ca(2+) release–activated Ca(2+) (CRAC) channels and store-operated Ca(2+) entry (SOCE), resulting in a disease syndrome called CRAC channelopathy that is characterized by severe dental enamel defects. The c...
שמור ב:
| הוצא לאור ב: | JCI Insight |
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| Main Authors: | , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Clinical Investigation
2017
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5358480/ https://ncbi.nlm.nih.gov/pubmed/28352661 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.91166 |
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