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Most brain disease-associated and eQTL haplotypes are not located within transcription factor DNase-seq footprints in brain
Dense genotyping approaches have revealed much about the genetic architecture both of gene expression and disease susceptibility. However, assigning causality to genetic variants associated with a transcriptomic or phenotypic trait presents a far greater challenge. The development of epigenomic reso...
Gorde:
| Argitaratua izan da: | Hum Mol Genet |
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| Egile Nagusiak: | , , , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
Oxford University Press
2017
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5351933/ https://ncbi.nlm.nih.gov/pubmed/27798116 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddw369 |
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