Silencing of human immunodeficiency virus long terminal repeat expression by an adenovirus E1a mutant.
Gene expression from the human immunodeficiency virus (HIV) long terminal repeat (LTR) is strongly stimulated by the viral tat gene. The HIV LTR is also activated by several physical and chemical agents and heterologous viral genes, including adenovirus E1a. As E1a has separable transcriptional acti...
Αποθηκεύτηκε σε:
| Εκδόθηκε σε: | Proc Natl Acad Sci U S A |
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| Κύριοι συγγραφείς: | , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
National Academy of Sciences
1990
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC53464/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2137611/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.87.4.1310 |
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