Silencing of human immunodeficiency virus long terminal repeat expression by an adenovirus E1a mutant.

Gene expression from the human immunodeficiency virus (HIV) long terminal repeat (LTR) is strongly stimulated by the viral tat gene. The HIV LTR is also activated by several physical and chemical agents and heterologous viral genes, including adenovirus E1a. As E1a has separable transcriptional acti...

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Detalhes bibliográficos

Publicado no:	Proc Natl Acad Sci U S A
Principais autores:	Ventura, A M, Arens, M Q, Srinivasan, A, Chinnadurai, G
Formato:	Artigo
Idioma:	Inglês
Publicado em:	National Academy of Sciences 1990
Assuntos:	Research Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC53464/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2137611/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.87.4.1310
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