Silencing of human immunodeficiency virus long terminal repeat expression by an adenovirus E1a mutant.

Gene expression from the human immunodeficiency virus (HIV) long terminal repeat (LTR) is strongly stimulated by the viral tat gene. The HIV LTR is also activated by several physical and chemical agents and heterologous viral genes, including adenovirus E1a. As E1a has separable transcriptional acti...

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Pubblicato in:	Proc Natl Acad Sci U S A
Autori principali:	Ventura, A M, Arens, M Q, Srinivasan, A, Chinnadurai, G
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	National Academy of Sciences 1990
Soggetti:	Research Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC53464/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2137611/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.87.4.1310
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