Accelerated Prion Disease in the Absence of Interleukin-10

The identity of pro- and anti-inflammatory cytokines in the neuropathogenesis of prion diseases remains undefined. Here we have investigated the role of anti-inflammatory cytokines on the progression of prion disease through the use of mice that lack interleukin-4 (IL-4), IL-10, IL-13, or both IL-4...

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Détails bibliographiques
Publié dans:J Virol
Auteurs principaux: Thackray, Alana M., McKenzie, Andrew N., Klein, Michael A., Lauder, Angus, Bujdoso, Raymond
Format: Artigo
Langue:Inglês
Publié: American Society for Microbiology (ASM) 2004
Sujets:
Pathogenesis and Immunity
Accès en ligne:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC533935/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15564479/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.78.24.13697-13707.2004
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