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TRIP13-deficient tubular epithelial cells are susceptible to apoptosis following acute kidney injury
Damage to renal tubular epithelial cells by genetic, environmental, or biological insults can initiate complex signaling mechanisms that promote kidney repair and functional recovery. In this study, we demonstrated that thyroid receptor interacting protein 13 (TRIP13) is a critical modulator of tubu...
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| 出版年: | Sci Rep |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5335694/ https://ncbi.nlm.nih.gov/pubmed/28256593 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep43196 |
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