Vasopressin type 1A receptor deletion enhances cardiac contractility, β-adrenergic receptor sensitivity and acute cardiac injury-induced dysfunction

Vasopressin type 1A receptor (V1AR) expression is elevated in chronic human heart failure (HF) and contributes to cardiac dysfunction in animal models, in part via reduced β-adrenergic receptor (βAR) responsiveness. Although cardiac V1AR overexpression and V1AR stimulation are each sufficient to dec...

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Detalhes bibliográficos
Publicado no:Clin Sci (Lond)
Main Authors: Wasilewski, Melissa A., Grisanti, Laurel A., Song, Jianliang, Carter, Rhonda L., Repas, Ashley A., Myers, Valerie D., Gao, Erhe, Koch, Walter J., Cheung, Joseph Y., Feldman, Arthur M., Tilley, Douglas G.
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5334456/
https://ncbi.nlm.nih.gov/pubmed/27589993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/CS20160363
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