Vasopressin type 1A receptor deletion enhances cardiac contractility, β-adrenergic receptor sensitivity and acute cardiac injury-induced dysfunction

Vasopressin type 1A receptor (V1AR) expression is elevated in chronic human heart failure (HF) and contributes to cardiac dysfunction in animal models, in part via reduced β-adrenergic receptor (βAR) responsiveness. Although cardiac V1AR overexpression and V1AR stimulation are each sufficient to dec...

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Vydáno v:Clin Sci (Lond)
Hlavní autoři: Wasilewski, Melissa A., Grisanti, Laurel A., Song, Jianliang, Carter, Rhonda L., Repas, Ashley A., Myers, Valerie D., Gao, Erhe, Koch, Walter J., Cheung, Joseph Y., Feldman, Arthur M., Tilley, Douglas G.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2016
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5334456/
https://ncbi.nlm.nih.gov/pubmed/27589993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/CS20160363
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