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Data supporting the inability of indomethacin to induce autophagy in U251 glioma cells
Autophagy, a catabolic process involving intracellular degradation of unnecessary or dysfunctional cellular components through the lysosomal machinery, could act as a prosurvival, as well as a cytotoxic mechanism (Parzych and Klionsky, 2014) [1]. Cyclooxygenase inhibitor indomethacin inhibits prolif...
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| Foilsithe in: | Data Brief |
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| Main Authors: | , , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Elsevier
2017
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5320059/ https://ncbi.nlm.nih.gov/pubmed/28243617 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.dib.2017.02.012 |
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