Prenatal alcohol exposure potentiates chronic neuropathic pain, spinal glial and immune cell activation and alters sciatic nerve and DRG cytokine levels

A growing body of evidence indicates that prenatal alcohol exposure (PAE) may predispose individuals to secondary medical disabilities later in life. Animal models of PAE reveal neuroimmune sequelae such as elevated brain astrocyte and microglial activation with corresponding region-specific changes...

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Detaylı Bibliyografya
Yayımlandı:Brain Behav Immun
Asıl Yazarlar: Noor, Shahani, Sanchez, Joshua J., Vanderwall, Arden G., Sun, Melody S., Maxwell, Jessie R., Davies, Suzy, Jantzie, Lauren L., Petersen, Timothy R., Savage, Daniel D., Milligan, Erin D.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2016
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5316367/
https://ncbi.nlm.nih.gov/pubmed/28011263
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbi.2016.12.016
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