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Synaptotagmin-1 and synaptotagmin-7-dependent fusion mechanisms target synaptic vesicles to kinetically distinct endocytic pathways
Synaptic vesicle recycling is essential for maintaining normal synaptic function. The coupling of exocytosis and endocytosis is assumed to be Ca(2+)-dependent but the exact role of Ca(2+) and its key effector synaptotagmin-1 (syt1) in regulation of endocytosis are poorly understood. Here, we probed...
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| Gepubliceerd in: | Neuron |
|---|---|
| Hoofdauteurs: | , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2017
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5300960/ https://ncbi.nlm.nih.gov/pubmed/28111077 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2016.12.010 |
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