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STAT3-targeted treatment with silibinin overcomes the acquired resistance to crizotinib in ALK-rearranged lung cancer
The signal transducer and activator of transcription 3 (STAT3) has been suggested to play a prominent role in mediating non–small-cell lung cancer (NSCLC) resistance to some tyrosine kinase inhibitor (TKI)-mediated therapies. Using a model of anaplastic lymphoma kinase gene (ALK)-translocated NSCLC...
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| Publicado no: | Cell Cycle |
|---|---|
| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Taylor & Francis
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5224449/ https://ncbi.nlm.nih.gov/pubmed/27753543 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2016.1245249 |
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