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VCP cooperates with UBXD1 to degrade mitochondrial outer membrane protein MCL1 in model of Huntington’s disease
Proteasome-dependent turnover of mitochondrial outer membrane (OMM)-associated proteins is one of the mechanisms for maintaining proper mitochondrial quality and function. However, the underlying pathways and their implications in human disease are poorly understood. Huntington’s disease (HD) is a f...
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| Veröffentlicht in: | Biochim Biophys Acta |
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| Hauptverfasser: | , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2016
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5219860/ https://ncbi.nlm.nih.gov/pubmed/27913212 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbadis.2016.11.026 |
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