Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture

Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of propensity to myocardial rupture after MI due to its...

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Bibliografske podrobnosti
izdano v:JCI Insight
Main Authors: Sutton, Nadia R., Hayasaki, Takanori, Hyman, Matthew C., Anyanwu, Anuli C., Liao, Hui, Petrovic-Djergovic, Danica, Badri, Linda, Baek, Amy E., Walker, Natalie, Fukase, Keigo, Kanthi, Yogendra, Visovatti, Scott H., Horste, Ellen L., Ray, Jessica J., Goonewardena, Sascha N., Pinsky, David J.
Format: Artigo
Jezik:Inglês
Izdano: American Society for Clinical Investigation 2017
Teme:
Research Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5213916/
https://ncbi.nlm.nih.gov/pubmed/28097233
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.89504
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