Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture

Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of propensity to myocardial rupture after MI due to its...

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Detalhes bibliográficos
Publicado no:JCI Insight
Main Authors: Sutton, Nadia R., Hayasaki, Takanori, Hyman, Matthew C., Anyanwu, Anuli C., Liao, Hui, Petrovic-Djergovic, Danica, Badri, Linda, Baek, Amy E., Walker, Natalie, Fukase, Keigo, Kanthi, Yogendra, Visovatti, Scott H., Horste, Ellen L., Ray, Jessica J., Goonewardena, Sascha N., Pinsky, David J.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2017
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5213916/
https://ncbi.nlm.nih.gov/pubmed/28097233
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.89504
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