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Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture
Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of propensity to myocardial rupture after MI due to its...
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| Veröffentlicht in: | JCI Insight |
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| Hauptverfasser: | , , , , , , , , , , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
American Society for Clinical Investigation
2017
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5213916/ https://ncbi.nlm.nih.gov/pubmed/28097233 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.89504 |
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