Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture

Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of propensity to myocardial rupture after MI due to its...

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Veröffentlicht in:JCI Insight
Hauptverfasser: Sutton, Nadia R., Hayasaki, Takanori, Hyman, Matthew C., Anyanwu, Anuli C., Liao, Hui, Petrovic-Djergovic, Danica, Badri, Linda, Baek, Amy E., Walker, Natalie, Fukase, Keigo, Kanthi, Yogendra, Visovatti, Scott H., Horste, Ellen L., Ray, Jessica J., Goonewardena, Sascha N., Pinsky, David J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Society for Clinical Investigation 2017
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Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5213916/
https://ncbi.nlm.nih.gov/pubmed/28097233
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.89504
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