Ectonucleotidase CD39-driven control of postinfarction myocardial repair and rupture

Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of propensity to myocardial rupture after MI due to its...

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Publicado en:JCI Insight
Autores principales: Sutton, Nadia R., Hayasaki, Takanori, Hyman, Matthew C., Anyanwu, Anuli C., Liao, Hui, Petrovic-Djergovic, Danica, Badri, Linda, Baek, Amy E., Walker, Natalie, Fukase, Keigo, Kanthi, Yogendra, Visovatti, Scott H., Horste, Ellen L., Ray, Jessica J., Goonewardena, Sascha N., Pinsky, David J.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Clinical Investigation 2017
Materias:
Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC5213916/
https://ncbi.nlm.nih.gov/pubmed/28097233
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.89504
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