Physiological Suppression of Lipotoxic Liver Damage by Complementary Actions of HDAC3 and SCAP/SREBP

Liver fat accumulation precedes non-alcoholic steatohepatitis, an increasing cause of end-stage liver disease. Histone Deacetylase 3 (HDAC3) is required for hepatic triglyceride homeostasis and Sterol Regulatory Element Binding Protein (SREBP) regulates the lipogenic response to feeding, but the cro...

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Detaylı Bibliyografya
Yayımlandı:Cell Metab
Asıl Yazarlar: Papazyan, Romeo, Sun, Zheng, Kim, Yong Hoon, Titchenell, Paul M., Hill, David A., Lu, Wenyun, Damle, Manashree, Wan, Min, Zhang, Yuxiang, Briggs, Erika R., Rabinowitz, Joshua D., Lazar, Mitchell A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2016
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5159233/
https://ncbi.nlm.nih.gov/pubmed/27866836
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2016.10.012
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