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Mutations in the Human AAA(+) Chaperone p97 and Related Diseases
A number of neurodegenerative diseases have been linked to mutations in the human protein p97, an abundant cytosolic AAA(+) (ATPase associated with various cellular activities) ATPase, that functions in a large number of cellular pathways. With the assistance of a variety of cofactors and adaptor pr...
Tallennettuna:
| Julkaisussa: | Front Mol Biosci |
|---|---|
| Päätekijät: | , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Frontiers Media S.A.
2016
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5131264/ https://ncbi.nlm.nih.gov/pubmed/27990419 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fmolb.2016.00079 |
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