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Hepatocyte ERBB3 and EGFR are required for maximal CCl(4)-induced liver fibrosis
Epidermal growth factor receptor (EGFR) and its ligands have been implicated in liver fibrosis. However, it has not been directly shown that hepatocellular genetic ablation of either this receptor tyrosine kinase or ERBB3, its interactive signaling partner, affects hepatic fibrosis. Carbon tetrachlo...
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| Publicado no: | Am J Physiol Gastrointest Liver Physiol |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5130544/ https://ncbi.nlm.nih.gov/pubmed/27586651 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00423.2015 |
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