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RAC1 GTPase promotes the survival of breast cancer cells in response to hyper-fractionated radiation treatment
Radiation therapy is a staple approach for cancer treatment, whereas radioresistance of cancer cells remains a substantial clinical problem. In response to ionizing radiation (IR) induced DNA-damage, cancer cells can sustain/activate pro-survival signaling pathways, leading to apoptotic resistance a...
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| Izdano u: | Oncogene |
|---|---|
| Glavni autori: | , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2016
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5112160/ https://ncbi.nlm.nih.gov/pubmed/27181206 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/onc.2016.163 |
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