Tunicamycin increases desensitization of acetylcholine receptors in cultured mouse muscle cells.

Whole-cell currents activated by acetylcholine (AcCho) were recorded in C2 mouse myotubes before and after prolonged treatment with tunicamycin, an inhibitor of glycosylation. In control cells the AcCho-induced currents decayed slowly even in the continuous presence of AcCho. After 24 hr of treatmen...

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Veröffentlicht in:	Proc Natl Acad Sci U S A
Hauptverfasser:	Giovannelli, A, Grassi, F, Eusebi, F, Miledi, R
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	National Academy of Sciences 1991
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC51114/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2000386/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.88.5.1808
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