Tunicamycin increases desensitization of acetylcholine receptors in cultured mouse muscle cells.
Whole-cell currents activated by acetylcholine (AcCho) were recorded in C2 mouse myotubes before and after prolonged treatment with tunicamycin, an inhibitor of glycosylation. In control cells the AcCho-induced currents decayed slowly even in the continuous presence of AcCho. After 24 hr of treatmen...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
1991
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC51114/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2000386/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.88.5.1808 |
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